An updated review of nicotine in gastrointestinal diseases / Pranay Wal (2023)  

Public Titre : An updated review of nicotine in gastrointestinal diseases Type de document : document électronique Auteurs : Pranay Wal, Auteur ; Namra Aziz, Auteur ; Aman Patel, Auteur ; Ankita Wal, Auteur Editeur : San Francisco [Etats-Unis] : Bentham Science Publishers Année de publication : 2023 Collection : The Open Public Health Journal, ISSN 1874-9445 num. 16(3) Importance : p. 1874-9445 Présentation : ill. Langues : Anglais (eng) Catégories : [TABAC] chimie du tabac:constituant:alcaloïde:nicotine [TABAC] étude [TABAC] tabagisme:pathologie:cancer [TABAC] tabagisme:pathologie:pathologie digestive [TABAC] tabagisme:risque Index. décimale : TA 3.2.2.3 Cancer Résumé : Background: Nicotine addiction is a major risk for the population all over the world. Many gastrointestinal diseases are caused due to nicotine addiction, like peptic ulcers, inflammatory bowel diseases, and gastrointestinal cancer. Nicotine causes variations in the offensive and defensive factors, which are involved in gastrointestinal mechanisms. Objective: We aim to provide insights into nicotine activity in gastrointestinal disorders and cancer. Methods: Extensive literature was done using the keywords “Nicotine,” “Nicotine addiction,” “GI disorders,” “Peptic ulcer,” “Inflammatory bowel diseases,” and “Gastrointestinal cancer” from standard databases like PubMed, Scopus, Elsevier, and Science Direct. Conclusion: Thus, we may conclude that addiction to nicotine in peptic ulcers causes an increase of offensive factors and a decrease of defensive factors, and in gastrointestinal cancers, causes an increase in angiogenesis, metastasis, and proliferation and decrease in apoptosis in ulcerative colitis. It is used as treatment but worsens the condition of Crohn’s disease. En ligne : https://doi.org/10.2174/0118749445271127231116130459 Format de la ressource électronique : Article en ligne Permalink : https://biblio.fares.be/opac_css/index.php?lvl=notice_display&id=10569

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Epidemiological evidence associating secondhand smoke exposure with cardiovascular disease / Brent E. Faught (2009)  

Public Titre : Epidemiological evidence associating secondhand smoke exposure with cardiovascular disease Type de document : texte imprimé Auteurs : Brent E. Faught, Auteur ; Andreas D. Flouris, Auteur ; John Cairney, Auteur Editeur : San Francisco [Etats-Unis] : Bentham Science Publishers Année de publication : 2009 Collection : Inflammation & allergy - drug targets, ISSN 1871-5281 num. 8 Importance : p. 321-327 Présentation : tab. Langues : Anglais (eng) Catégories : [TABAC] étude:épidémiologie [TABAC] tabagisme:évaluation du tabagisme:marqueur [TABAC] tabagisme:pathologie:pathologie cardio-vasculaire [TABAC] tabagisme:tabagisme passif:tabagisme environnemental Index. décimale : TA 7.4.2.2 chez l'adulte Résumé : The objective of this paper was to review the epidemiological literature examining the association between secondhand smoke (SHS) and cardiovascular disease (CVD). Specifically, we examined the various screening methods available in assessing smoking behaviour and quantifying nicotine absorption. Further, we considered the natural history of those exposed to SHS and the associated risk of CVD. We reviewed routine methods used to assess exposure to SHS; evaluated the utility of subjective screening questions regarding smoking behaviour and examined the efficacy of nicotine and cotinine biomarkers used to quantify SHS exposure in epidemiological and clinical-based research. Self-reporting is practical and cost-effective in identifying smoking behaviour patterns, but is subject to recall bias and underestimation of exposure, especially in the presence of children. Nicotine and cotinine biomarkers have proven valuable in quantifying tobacco smoke absorption and establishing biological plausibility. A combination of SHS self-reported and biomarker evaluation provide the most stringent method of establishing exposure. Sufficient evidence is reported in epidemiological research to support a causal association between SHS exposure and increased risks of CVD morbidity and mortality among both men and women. The risk of developing an acute cardiac syndrome or chronic lifetime coronary events is at least 30%. Similarly, reduction in the incidence of a myocardial infarction decreases by nearly 50% in the absence of SHS. Considering the biological plausibility and dose-response relationship between SHS and CVD, effective interventions that incorporate a comprehensive screening method of behavioral and biological measures of exposure coupled with efficacious treatment should elicit favorable change for at-risk populations. En ligne : http://dx.doi.org/10.2174/1871528110908050321 Format de la ressource électronique : PDF Permalink : https://biblio.fares.be/opac_css/index.php?lvl=notice_display&id=8056

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